Amygdala Neuroplasticity and Pain Behaviors with a Focus on CRF Neurons

Neuroplasticity in the central amygdala is causally linked to pain behaviors in different models. Electrophysiological approaches for the analysis of neural activity changes in pain conditions will be discussed. Pharmacological, optogenetic and chemogenetic techniques will be presented to link neural activity and neuroplastic changes to pain behaviors. A focus will be on CRF neurons.